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March 22, 2020: by Bill Sardi
A newly published report indicates beta cyclodextrin, a solubilizing and anti-cholesterol agent in Longevinex®, inhibits attachment of coronaviruses to infect cells. According to a report published in the March 2020 issue of Acta Biomedica, cyclodextrin also inhibits the molecular doorway (ACE2) for coronaviruses to enter cells. Cyclodextrin essentially competes with viral targets for entry into healthy cells. Cyclodextrins are sugar-like molecules that serve as stabilizers and as a molecular delivery system.
Longevinex® Provides Five Molecules That Either Inhibit Coronavirus Infection Or Support Overall Immunity
Acta Biomed. 2020 Mar 19;91(1):161-164. doi: 10.23750/abm.v91i1.9402.
Viral infectivity depends on interactions between components of the host cell plasma membrane and the virus envelope. Here we review strategies that could help stem the advance of the SARS-COV-2 epidemic.
We focus on the role of lipid structures, such as lipid rafts and cholesterol, involved in the process, mediated by endocytosis, by which viruses attach to and infect cells. Previous studies have shown that many naturally derived substances, such as cyclodextrin and sterols, could reduce the infectivity of many types of viruses, including the coronavirus family, through interference with lipid-dependent attachment to human host cells.
Certain molecules prove able to reduce the infectivity of some coronaviruses, possibly by inhibiting viral lipid-dependent attachment to host cells. More research into these molecules and methods would be worthwhile as it could provide insights the mechanism of transmission of SARS-COV-2 and, into how they could become a basis for new antiviral strategies.
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